Job Watch

HEAL initiative: Pharmacotherapies to Reverse Opioid Overdose Induced Respiratory Depression Without Central Opioid Withdrawal (Target Validation and Candidate Therapeutic Development) (UG3/UH3 Clinical Trials Not Allowed)

Funding Opportunity RFA-HL-22-013 from the NIH Guide for Grants and Contracts. This Funding Opportunity Announcement (FOA) aims to stimulate the pre-clinical development of novel, pharmacotherapeutic candidates to selectively counteract opioid induced respiratory depression (OIRD), without blocking opioid analgesic benefits or inducing acute opioid withdrawal.

Review of the Accuracy of Grants Information for Fiscal Year 2021

Notice NOT-OD-21-186 from the NIH Guide for Grants and Contracts

Advancing Gender Inclusive Excellence (AGIE) Coordinating Center (U54 Clinical Trial Not Allowed)

Funding Opportunity RFA-OD-21-010 from the NIH Guide for Grants and Contracts. The purpose of this funding opportunity announcement (FOA) is to solicit applications for a Coordinating Center (U54) to serve as a resource hub for existing and future Advancing Gender Inclusive Excellence (AGIE) programs. The AGIE Coordinating Center will provide the organizational framework for the management, direction, and overall coordination of all common activities. Specifically, this program is aimed at investigating strategies, approaches, barriers, and interventions to women attaining leadership positions in many areas of science, technology, engineering.

Role of Astrocytes in Degeneration of the Neurovascular Unit in AD/ADRDs (R01 Clinical Trial Not Allowed)

Funding Opportunity PAR-22-037 from the NIH Guide for Grants and Contracts. The neurovascular unit involves multiple pathways that contribute to neurodegeneration. Astrocytes, due to their overlapping roles regulating the blood brain barrier, neuronal health and response to degenerating cells, are uniquely positioned to be therapeutic targets. Astrocytes are a fundamental component of the neurovascular unit and are known to play a role in regulating the blood brain barrier and APOE signaling. However, the mechanistic role of astrocytes for the neurovascular unit in health and disease, including in vascular contributions to cognitive impairment and dementia (VCID) and across the spectrum of AD/ADRD diagnoses, is largely unknown, and represents a gap area and an opportunity for research investment. This initiative would represent the first targeted initiative on the fundamental role of astrocytes in AD/ADRD at the NIH.

RESEARCH SPECIALIST - University of Wisconsin–Madison - Madison, WI

Indeed.com - Bioinformatics - Wed, 2021-09-29 14:57
This position will include working animal husbandry, cell culture, DNA / RNA extraction, qPCR, CRISPR assays and basic cloning, and fluorescent microscopy…
From University of Wisconsin–Madison - Wed, 29 Sep 2021 18:57:24 GMT - View all Madison, WI jobs
Categories: Job Watch

Limited Competition: Coordinating Center (CC) for the Small Cell Lung Cancer (SCLC) Consortium (U24 Clinical Trial Not Allowed)

Funding Opportunity PAR-21-346 from the NIH Guide for Grants and Contracts. The purpose of this limited competition Funding Opportunity Announcement (FOA) is to continue support for the Coordinating Center (CC) for the Small Cell Lung Cancer (SCLC) Consortium (Consortium) that will be focused on prevention, diagnosis, treatment, and mechanisms of treatment resistance in SCLC. The CC will support the administrative coordination of the SCLC Consortium, create and support database(s) for -omics or other data pertinent to the Consortium, secure centralized tissue banking for specimens submitted by the members of the Consortium and a virtual biospecimen database that would include all tissue resources of the Consortium, provide centralized biostatistics, bioinformatics and data analysis support, establish SCLC in vivo and in vitro model repositories and distribution units, support meeting and communications coordination among members of Consortium, and form and maintain a Consortium website.

NIA Career Transition Award (K22 Independent Clinical Trial Not Allowed)

Funding Opportunity PAR-21-351 from the NIH Guide for Grants and Contracts. The purpose of the NIA Career Transition Award (CTA) is to facilitate the transition of mentored researchers to tenure-track faculty conducting research that advances the mission of NIA. This three-year award provides protected time through salary and research support and is targeted at applicants who plan to start a tenure-track faculty position within a year of the award.

Interventions for Stigma Reduction to Improve HIV/AIDS Prevention, Treatment and Care in Low- and Middle- Income Countries (R01 - Clinical Trial Optional)

Funding Opportunity PAR-21-344 from the NIH Guide for Grants and Contracts. To develop and/or pilot test interventions for HIV/AIDS-associated stigma and its outcome on the prevention and treatment of HIV/AIDS and on the quality of life of People Living with HIV/AIDS (PLWH). Specifically, this initiative will support research on or leading to interventions to address a) innovation in measurement of HIV-associated stigma and of other intersecting stigmas due to multiple morbidities to develop better interventions, b) stigma and adolescent and/or youth health, c) effects of stigma on family members or care givers of PLWH, and on the aging PLWH, d) novel stigma reduction interventions that link to increase in care-seeking behavior and/or decrease in transmission and e) coping with the complexity of added burden of stigmatization due to HIV and to one or more comorbidities/coinfections.

NINDS Institutional AD/ADRD Research Training Program (T32 Clinical Trial Not Allowed)

Funding Opportunity PAR-22-021 from the NIH Guide for Grants and Contracts. The purpose of this FOA is to provide support for institutional research training programs in Alzheimers Disease/Alzheimers Disease-Related Dementias (AD/ADRD). These institutional research training programs should produce well-trained neuroscientists who leave the program with the research skills and scientific knowledge to make a significant contribution to research on AD/ADRD cognitive impairment and dementia. Programs should be designed to enhance the breadth and depth of training across the spectrum AD/ADRD research areas (e.g. AD, Vascular Contributions to Cognitive Impairment and Dementia (VCID), Lewy Body Dementia (LBD), Fronto-temporal Dementia (FTD) and mixed dementias) by incorporating didactic, research and career development components within this theme into a program that fosters exceptional research skills and knowledge. Programs may support basic, clinical and/or translational research. Programs supported by this FOA must include formal components to ensure a thorough understanding of experimental design, statistical principles and methodological approaches, analytical skills, and skills for communicating science, both orally and in writing, to a wide variety of audiences. All programs are expected to design and/or provide opportunities and activities that will foster the development of quantitative literacy and the application of quantitative approaches to the trainees' research. These training programs are intended to be 2 years in duration and support training of one or more of the following groups: dissertation stage predoctoral students in their 3rd and/or 4th year of graduate school, postdoctoral fellows and fellowship-stage clinicians. (NINDS does not support first or second year graduate students under this FOA).

The role of Epstein Barr virus (EBV) infection in Non-Hodgkin Lymphoma (NHL) and Hodgkin disease (HD) development with or without an underlying HIV infection (U01 Clinical Trial Optional)

Funding Opportunity PAR-21-348 from the NIH Guide for Grants and Contracts. The role of EBV infection on Non-Hodgkin Lymphoma (NHL) and Hodgkin disease (HD) development with or without an underlying HIV infection and AIDS will increase our understanding through mechanistic, epidemiological, or translational studies that examine how EBV promotes NHL or HD initiation, progression and the resulting disease sequelae. In addition, insights into mechanistic differences of EBV infection and lymphomagenesis between HIV positive and HIV negative persons will be gained.

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